You got published? Awesome! Congratulations.
Were you also the one Trump tweeted about in reference to covid or am I thinking of someone else?
That's a funny story. In early 2020, I did a number of writeups summarizing COVID-19 pathology papers (a lot of my detractors on SB/SV/QQ whined that I was doing "epidemiology", when the spread of the disease wasn't really my focus at all; I was mostly digging into papers on COVID-19 pathology and proteomics, as in, what the virus specifically does to the body).
Michael Coudrey, a.k.a. "Mike Tokes", a friend of Baked Alaska, made a thread about my posts. Then, Trump retweeted him. Then, media watchdog groups flipped out. "Oh my god, who is @b00msl4ang and why is Trump retweeting them as a source?"
The comments are a hilarious time capsule. Note how many frothing anti-Trumpists there are in the comments saying the virus was completely harmless just because Mike Tokes was saying it was harmful. They all changed their tune completely in a couple months. "Fauci, lock us down more!"
Huh, that's interesting. Let's see if the claims in that thread still hold up.
There are two lineages, L and S
Way more than that, now.
The disease has an R0 of around 6.6
By early calculations, yes. Now? They estimate that it's lower, but still exponential.
The median incubation period is around 5 days, but outliers of 24+ days have been seen
Those outliers proved very rare indeed. It is mostly around 2 to 5 days.
SARS-CoV-2 is spreading at alarming levels due to its ability to be airborne
It absolutely was airborne and the WHO denied it and said it was droplet-borne for the better part of a year (hence the useless interventions like surface decontamination and Plexiglas barriers).
SARS-CoV-2 is capable of asymptomatic transmission and it is practically impossible to stop the spread by contact tracing and minor quarantines
The infectiousness of asymptomatic carriers turned out to be much lower than they thought. Someone basically needs to be coughing and hacking and sweating to exude enough aerosols to do anything.
SARS-CoV-2 can enter the eye through the ocular surface
Potentially, yes, but it prefers the airway.
The virus attacks ACE2 receptors in cells
Without a single doubt.
This system is essential for your body to maintain the correct blood pressure
The RAAS is absolutely vital for maintaining blood pressure and volume, yes. That's why ACE2 is all over the body, and it's why the virus had a tropism for such a wide array of tissues (COVID toes, anyone?).
ACE2 receptors are found in the seminiferous ducts of the testis
They are. And the vascular endothelium, brain tissue, liver, pancreas, kidneys, heart, bile ducts, intestines, et cetera. In fact, a common early manifestation of COVID-19 is gastroenteritis-like symptoms.
SARS-CoV-2 can potentially cause lung fibrosis and chronic lung disease if not caught and treated with antivirals at an early stage
SARS did this too. It's actually caused by your own immune cells; monocyte infiltration of the lungs, to be specific. The lung tissue just starts fibrosing and scarring over out of the blue.
SARS-CoV (a relative of SARS-CoV-2) has been shown to cause severe neural death (surprisingly without encephalitis) in transgenic mouse models
SARS-CoV-2 has now been shown to productively infect both astrocytes and microglia and be capable of both retrograde and anterograde movement along nervous tissue. It is absolutely neurotropic.
Recent information seems to suggest that SARS-CoV-2 can cause neurological symptoms and cerebrovascular disease, leading to loss of autonomic functions of the brain and, in the worst case, lingering brain damage
Reports of Post-COVID "Brain Fog" and dysautonomia are common, but actual severe COVID-19 encephalitis is rare.
This could cause a significant portion of the population to have severe drops in cognitive function, intelligence, and IQ
This was completely and utterly vindicated, with multiple papers now stating that COVID-19 can cause an IQ loss of up to half a standard deviation. It's like exposing the brain to decades of aging.
The action of SARS-Coronaviruses (which attack ACE2 pathways) can dysregulate the angiotensin system and cause cardiopulmonary damage and inflammation directly through this route
Potentially, yes. The RAAS is involved in inflammatory responses.
SARS-CoV-2 can cause myocarditis leading to myoglobin accumulation in the blood and renal failure
Early reports of COVID-19-induced myocarditis were met with follow-up studies where doctors were actually bewildered by the
lack of heart injury due to COVID-19 infections, given its prevalence in early reports out of China. Were they priming people to treat COVID-19 vaccine-induced myocarditis as having a viral origin?
What disturbs me the most about this virus is that it can potentially target so many different kinds of tissues
It can.
An autopsy, may reveal hardly any lung pathology whatsoever
Actually very rare. It primarily affects the lungs.
There are so many different things that could cause the collapses we have witnessed
The collapses and seizures seen in early footage out of China were not matched elsewhere. Very bizarre in retrospect. How many of those early vids were psyops?
We still have the archived footage.
However, what I found most concerning about the collapses is the consistent presentation of the victim ... These people, their limbs stretch out straight, and they begin to seize or show the fencing sign
The thing is, a lot of those early vids of people collapsing in the street
do show abnormal posturing indicative of brain damage.
Coronavirus does not just effect the respiratory system, it embeds itself into the central nervous system & has the potential to cause significant neurological impairment, brain damage, & cancer
Potentially, yes.
All in all, most of my claims in early 2020
did hold up.
