I thought the same of China and now Corona Chan has arrived. Something will give and new players will seize that opportunity to reshuffle the board. Who knows if China will lose some land or two.
SUMMARY OF SARS-CoV/SARS-CoV-2 AND COVID-19 FINDINGS SECTION I: Overview Current COVID-19 tracking services: Johns Hopkins New York Times (NYT) Worldometer Evaluation of current therapies: Chloroquine: Showing the most promise Hydroxychloroquine: Effective, but many side effects including pro...
docs.google.com
This is what the virus actually does to the human body, according to everything I've dug up on it:
Bilateral viral pneumonia and ARDS.
Lung fibrosis and permanent loss of lung function.
Myocarditis and pericarditis and the breakdown of the heart muscle into the bloodstream, accompanied by elevated troponin levels.
Excessive clotting leading to myocardial infarction, pulmonary embolism, or stroke. Platelets are used up by what appears to be Disseminated Intravascular Coagulation.
Mild viral hepatitis, indicated by abnormal AST/ALT and highly elevated ferritin. Myoglobinemia and possible renal tubular damage that leads to acute kidney injury.
Cytokine release syndrome.
Bacterial co-infections and viral sepsis leading to multiple organ failure and excess clotting.
Hypokalemia, low lymphocytes, low platelets, and low white blood cell counts.
Male infertility and damage to the seminiferous ducts, indicated by abnormal testosterone and luteinizing hormone levels.
Medullary damage that causes dysautonomia, loss of automatic breathing, and perhaps even collapses and seizures with abnormal posturing (decerebrate/decorticate posturing) and the fencing response. Behavioral abnormalities like strange aggression have also been noted.
Possible systemic hypertension, vasculitis, and/or hemolytic anemia.
People need to get mad about this fucking virus, and they need to speak up.
Yep. Myocarditis and pericarditis. COVID-19 dissolves the heart. Many people dying in the ICU are dying of cardiomyopathy, not pneumonia.
The ventilators are useless, by the way.
They're pumping air into critical COVID-19 patients' lungs to try and inflate their alveoli and deal with the pneumonia, right? Well, the problem isn't one of pneumonia and collapsed alveoli.
Patients have low blood O2 because the virus actually damages blood cells and blood vessels. It's less of a lung disease and more of a blood disease.
The novel coronavirus pneumonia (COVID-19) is an infectious acute respiratory caused by the novel coronavirus. The virus is the positive-strand RNA one with high homology to bat coronavirus. The pathogenic mechanism of the new coronavirus is still unclear, which is a significant obstacle to the...
chemrxiv.org
SARS was known to cause systemic vasculitis, so why not COVID-19?
In order to investigate the clinical pathology of severe acute respiratory syndrome (SARS), the autopsies of three patients who died from SARS in Nan Fang Hospital Guangdong, China were studied retrospectively. Routine haematoxylin and eosin (H&E) staining was used to study all of the tissues fr …
www.ncbi.nlm.nih.gov
This is also why the virus causes a fuck-ton of clotting and blood clots roaming around that can cause strokes, heart attacks, and PE.
It's because the sepsis and free heme cause coagulation.
Jesus Christ, I am sick to death of doctors being sent into this shit thinking "it's just pneumonia", because of improper briefings that don't point out the multi-systemic inflammation of SARS-like viruses.
This is what the virus actually does to the human body, according to everything I've dug up on it:
Bilateral viral pneumonia and ARDS.
Lung fibrosis and permanent loss of lung function.
Myocarditis and pericarditis and the breakdown of the heart muscle into the bloodstream, accompanied by elevated troponin levels.
Excessive clotting leading to myocardial infarction, pulmonary embolism, or stroke. Platelets are used up by what appears to be Disseminated Intravascular Coagulation.
Mild viral hepatitis, indicated by abnormal AST/ALT and highly elevated ferritin. Myoglobinemia and possible renal tubular damage that leads to acute kidney injury.
Cytokine release syndrome.
Bacterial co-infections and viral sepsis leading to multiple organ failure and excess clotting.
Hypokalemia, low lymphocytes, low platelets, and low white blood cell counts.
Male infertility and damage to the seminiferous ducts, indicated by abnormal testosterone and luteinizing hormone levels.
Medullary damage that causes dysautonomia, loss of automatic breathing, and perhaps even collapses and seizures with abnormal posturing (decerebrate/decorticate posturing) and the fencing response. Behavioral abnormalities like strange aggression have also been noted.
Possible systemic hypertension, vasculitis, and/or hemolytic anemia.
Did you manage to get your percentages and probabilities recorded? Because if this happens in like 0.002% of cases then that is different to it happening in say 40% of cases.
Did you also determine what qualifiers there were for how and why some people acquire additional symptoms or the virus takes a different route. Are we talking co-morbidities, age, genetics, lifestyle choices, strains, duration or intensity of exposure?
These are the questions that really need answers and that can yield practical information people can act upon. Without numbers it isn't something people can act on
I would recommend focusing on those factors to present a full picture. What you have is a collection of information but it is out of context, you need figures so people can understand it better.
Data is one thing, but the application of that data is equally if not more important. People need to know how to apply the information to themselves, that is the key step. And for that you need something universal that all can relate to, nothing is more universal than numbers.
If you really want to make it applicable and genuinely useful, you know what to do
If there are known complications and co-morbidities then someone somewhere must have had them, or else they would not have the data in the first place. So that is one case at least reported, studied, classified. If there have been no cases of these complications and you are merely speculating based on performance by related virus performance, then your data is extremely flawed and you should not be presenting it as fact, just speculation by a layman.
For the record I took college level chemistry, I'm no genius but I do have a working understanding of how this stuff works. I couldn't speculate without a lot more data and my good friend who does work in actual biochemistry fields also wouldn't chance a prediction on percentages when I asked a few weeks back.
You need to find those numbers. They must exist because I found some basic stats when I went and googled. Get some figures man, if you can't find them for this illness look at prior ones as your baseline
Actually, the latest speculation I heard is that it wasn't engineered, but rather that a researcher at the Institute of Virology or whatever it's called in Wuhan fucked up and got the virus. Then managed to go about merrily passing it around until things took a turn for the worst. The institute was actually working on bat viruses because they're known to be a huge reservoir of disease, and the closest matches originate several hundred miles away from Wuhan.
I'd have to go back and find the details, but the researcher in question hasn't been seen in public since the suggestions surfaced, yet the Chinese have been denying the researcher had anything to do with it (and claim she's still alive but, oddly, haven't produced her at a news conference to shoot that claim down).
I think him and other Far Lefties associated with China are kinda holding onto the idea that China’s invincible and the way to the future together with Globalism
They’re hoping their status quo and worldview doesn’t change, otherwise they have to deal with the fact that they made some very deadly decisions
I think they're political fellow travelers, actually, not just that the belief they need to suck up to China. It is good in the sense that it's exposing the rot for all to see, though. Sunshine does tend to do that.
I thought the hypoxia would simply be caused by the combination of the virus killing cells in the lungs, and the histamine response causing too much mucus.
Actually, the latest speculation I heard is that it wasn't engineered, but rather that a researcher at the Institute of Virology or whatever it's called in Wuhan fucked up and got the virus. Then managed to go about merrily passing it around until things took a turn for the worst. The institute was actually working on bat viruses because they're known to be a huge reservoir of disease, and the closest matches originate several hundred miles away from Wuhan.
I'd have to go back and find the details, but the researcher in question hasn't been seen in public since the suggestions surfaced, yet the Chinese have been denying the researcher had anything to do with it (and claim she's still alive but, oddly, haven't produced her at a news conference to shoot that claim down).
I thought the hypoxia would simply be caused by the combination of the virus killing cells in the lungs, and the histamine response causing too much mucus.
Pretty much, that would be the most common factor.
A study that Dodger linked suggests there could be issues with the virus affecting red blood cell production, however that was a computer based model and not an actual clinical study so it is theory, not fact.
Actually, the latest speculation I heard is that it wasn't engineered, but rather that a researcher at the Institute of Virology or whatever it's called in Wuhan fucked up and got the virus. Then managed to go about merrily passing it around until things took a turn for the worst. The institute was actually working on bat viruses because they're known to be a huge reservoir of disease, and the closest matches originate several hundred miles away from Wuhan.
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docs.google.com
chemrxiv.org
www.ncbi.nlm.nih.gov
www.westernjournal.com
